Search results for "Antidepressive Agent"

showing 10 items of 191 documents

Macrophage migration inhibitory factor is critically involved in basal and fluoxetine-stimulated adult hippocampal cell proliferation and in anxiety,…

2011

Intensive research is devoted to unravel the neurobiological mechanisms mediating adult hippocampal neurogenesis, its regulation by antidepressants, and its behavioral consequences. Macrophage migration inhibitory factor (MIF) is a pro-inflammatory cytokine that is expressed in the CNS, where its function is unknown. Here, we show, for the first time, the relevance of MIF expression for adult hippocampal neurogenesis. We identify MIF expression in neurogenic cells (in stem cells, cells undergoing proliferation, and in newly proliferated cells undergoing maturation) in the subgranular zone of the rodent dentate gyrus. A causal function for MIF in cell proliferation was shown using genetic (M…

Receptors SteroidStem-Cellsanimal diseasesmedicine.medical_treatmentHippocampusExpressionHippocampal formationHippocampusSubgranular zonememoryMice0302 clinical medicineConditioning PsychologicalCyclin D2Rat Dentate GyrusMice KnockoutNeurons0303 health sciencesMicroscopy ConfocalChronic StressMifNeurogenesisBrainFearrespiratory systemanxietyPsychiatry and Mental healthC-Reactive ProteinCytokinemedicine.anatomical_structuredepressionAntidepressive Agents Second-GenerationStem cellPsychologyAnimal-ModelNeurogenesisSpatial BehaviorNerve Tissue Proteinschemical and pharmacologic phenomena03 medical and health sciencesCellular and Molecular Neurosciencemedicineotorhinolaryngologic diseasesAnimalsRats WistarMaze LearningMacrophage Migration-Inhibitory FactorsMolecular BiologyCell Proliferation030304 developmental biologyMemory DisordersDentate gyrusfluoxetineFactor Mifbiological factorsRatsDisease Models AnimalAcoustic StimulationBromodeoxyuridineMacrophage migration inhibitory factorCorticosteroneNeuroscience030217 neurology & neurosurgery
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The neurobiological bases for the pharmacotherapy of nicotine addiction.

2007

Nicotine, the major psychoactive agent present in tobacco, acts as a potent addictive drug both in humans and laboratory animals, whose locomotor activity is also stimulated. A large body of evidence indicates that the locomotor activation and the reinforcing effects of nicotine may be related to its stimulatory effects on the mesolimbic dopaminergic function. Thus, it is now well established that nicotine can increase in vivo DA outflow in the nucleus accumbens and the corpus striatum. The stimulatory effect of nicotine on DA release most probably results from its ability to excite the neuronal firing rate and to increase the bursting activity of DA neurons in the substantia nigra pars com…

RAT STRIATAL SYNAPTOSOMESNicotineINDUCED BEHAVIORAL SENSITIZATIONmedia_common.quotation_subjectSubstantia nigraStriatumNicotinic AntagonistsBiologyNucleus accumbensPharmacologyReceptors NicotinicNicotineDrug DiscoverySUSTAINED-RELEASE BUPROPIONmedicineLOCOMOTOR STIMULANT ACTIONAnimalsHumansNicotinic Agonistsmedia_commonPharmacologyMIDBRAIN DOPAMINE NEURONSPars compactaAddictionNIGRA PARS COMPACTAFACILITATES SMOKING CESSATIONTobacco Use DisorderSUBUNIT MESSENGER-RNAAntidepressive AgentsVentral tegmental areaVENTRAL TEGMENTAL AREANicotinic agonistmedicine.anatomical_structurenervous systemmedicine.drugSEROTONIN(2C) RECEPTORS BLOCKSCurrent pharmaceutical design
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Early improvement as a resilience signal predicting later remission to antidepressant treatment in patients with Major Depressive Disorder: Systemati…

2017

Early improvement of depressive symptoms during the first two weeks of antidepressant treatment has been discussed to be a resilience signal predicting a later positive treatment outcome in patients with Major Depressive Disorder (MDD). However, the predictive value of early improvement varies between studies, and the use of different antidepressants may explain heterogeneous results. The objective of this review was to assess the predictive value of early improvement on later response and remission and to identify antidepressants with the highest chance of early improvement. We included 17 randomized controlled trials investigating early improvement in 14,779 adult patients with MDD compar…

medicine.medical_specialtymedicine.drug_classmedia_common.quotation_subjectMirtazapineTricyclic antidepressantPlacebolaw.invention03 medical and health sciences0302 clinical medicineRandomized controlled triallawInternal medicineOutcome Assessment Health CaremedicineHumansPsychiatryBiological Psychiatrymedia_commonDepressive Disorder MajorResilience Psychologicalmedicine.diseaseAntidepressive Agents030227 psychiatryPsychiatry and Mental healthMeta-analysisAntidepressantMajor depressive disorderPsychological resiliencePsychology030217 neurology & neurosurgerymedicine.drugJournal of psychiatric research
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Locomotor and antidepressant-like effects of 5-HT(1A) agonist LY 228729 in prenatally benzodiazepine-exposed rats.

1998

Locomotor activity and antidepressant-like effect in the forced swim test (FST) of 5-HT(1A) agonist LY 228729 were investigated in adult rats prenatally exposed at doses of diazepam (DZ) and alprazolam (ALP) which induce persistent downregulation of GABA/ benzodiazepine (BZ) receptors. Prenatal exposure to ALP and DZ did not modify the efficacy of subchronic LY 228729 to decrease immobility time in the FST. Prenatal DZ and ALP potentiated the facilitatory effect of subchronic LY 228729 on locomotor activity; prenatal DZ was more effective than prenatal ALP. Moreover, prenatal DZ increased stereotypic movements induced by LY 228729. These data suggest that the persistent downregulation of GA…

AgonistMalemedicine.drug_classInjections SubcutaneousPharmacologyMotor ActivityRats Sprague-DawleyDownregulation and upregulationPregnancymedicineAnimalsPharmacology (medical)ErgolinesReceptorBiological Psychiatry5-HT receptorSwimmingPharmacologyBenzodiazepineDepressive DisorderDiazepamAlprazolamChemistrymusculoskeletal neural and ocular physiologyAntidepressive AgentsRatsSerotonin Receptor AgonistsPsychiatry and Mental healthNeurologyAlprazolamAnti-Anxiety AgentsPrenatal Exposure Delayed EffectsFemaleNeurology (clinical)Stereotyped Behaviorhuman activitiesDiazepammedicine.drugBehavioural despair testEuropean neuropsychopharmacology : the journal of the European College of Neuropsychopharmacology
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A common role for psychotropic medications: memory impairment.

2002

Summary The psychopathologic profile of mental disorders is very diverse and psychotropic medications used to treat them differ in their chemical structure. Nevertheless, these drugs share these four characteristics: delayed onset of clinical response, not one of them can be said to cure, there is a high number of non-responders, and the mechanism responsible for their therapeutic action is not known. It is hypothesized that the action of psychotropic medications is memory impairment, understanding memory as the trace left in the nervous system not only by individual experiences but also by genetic and epigenetic phenomena. It is suggested that it would be beneficial to translate some resea…

medicine.medical_specialtyPsychotropic DrugsTherapeutic actionMechanism (biology)Mental DisordersDelayed onsetBrainGeneral MedicineModels PsychologicalAntidepressive AgentsAction (philosophy)Research strategiesMemorymedicineCyclic AMPMemory impairmentHumansPsychologyPsychiatryClinical psychologyAntipsychotic AgentsSignal TransductionMedical hypotheses
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Why do antidepressant therapies have such a poor success rate?

2016

Major depression is one of the leading global diseases [1,2]. Treatment is required, because untreated patients suffer not only from impaired emotion [3,4], but also from cognitive [4–6] and somati...

medicine.medical_specialtybusiness.industryDepressionGeneral NeuroscienceCognitionAntidepressive Agents030227 psychiatryPsychotherapy03 medical and health sciences0302 clinical medicineText miningMedicineAntidepressantHumansPharmacology (medical)Neurology (clinical)businessPsychiatry030217 neurology & neurosurgeryDepression (differential diagnoses)Expert review of neurotherapeutics
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Serotonin, the Prefrontal Cortex, and the Antidepressant-Like Effect of Cannabinoids

2007

Preclinical and clinical studies show that cannabis modulates mood and possesses antidepressant-like properties, mediated by the agonistic activity of cannabinoids on central CB1 receptors (CB1Rs). The action of CB1R agonists on the serotonin (5-HT) system, the major transmitter system involved in mood control and implicated in the mechanism of action of antidepressants, remains however poorly understood. In this study, we demonstrated that, at low doses, the CB1R agonist WIN55,212-2 [R(+)-[2,3-dihydro-5-methyl-3-[(morpholinyl)]pyrrolo[1,2,3-de]-1,4-benzoxazinyl]-(1-naphthalenyl) methanone mesylate] exerts potent antidepressant-like properties in the rat forced-swim test (FST). This effect …

MaleSerotoninJournal ClubMorpholinesmedicine.medical_treatmentPrefrontal CortexNaphthalenesPharmacologyEuphoriantAntidepressant likeRats Sprague-Dawleychemistry.chemical_compoundReceptor Cannabinoid CB1mental disordersAnimalsEthanolamideMedicineReceptorPrefrontal cortexNeuronsCannabinoidsDepressionbusiness.industryorganic chemicalsGeneral NeuroscienceAnandamideAntidepressive AgentsBenzoxazinesRatschemistryCannabinoidSerotoninbusinessNeuroscienceThe Journal of Neuroscience
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Antidepressant and anxiolytic effects of alprazolam versus the conventional. antidepressant desipramine and the anxiolytic diazepam in the forced swi…

1992

The antidepressant and anxiolytic effects of alprazolam were compared to those of desipramine, diazepam and buspirone in the forced swim test. Subchronic alprazolam induced a reduction in immobility similar to that of desipramine in 'non-pretested' and 'pretested' rats. In 'non-pretested' rats, the anti-immobility effect of desipramine was potentiated by diazepam and alprazolam, given before subchronic desipramine, while the anti-immobility effect of subchronic alprazolam was counteracted by diazepam. Diazepam, administered before the pretest session, counteracted, 24 h later, the anti-immobility effect of subchronic desipramine and alprazolam; alprazolam counteracted the anti-immobility ef…

Malemedicine.drug_classPharmacologyAnxiolyticBuspironeDesipraminemedicineAnimalsSwimmingPharmacologyBenzodiazepineDiazepamAlprazolamDepressionDesipramineRats Inbred StrainsReceptors GABA-AAntidepressive AgentsBuspironeRatsAnti-Anxiety AgentsAlprazolamReceptors SerotoninAntidepressantPsychologyDiazepamBehavioural despair testmedicine.drugEuropean Journal of Pharmacology
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Impact of the WHO depression guideline on patient care by psychiatrists: a randomized controlled trial.

2008

AbstractBackgroundScientific literature reviews aim to summarize the state of knowledge and published empirical evidence. In contrast, medical guidelines are intervention tools that aim to improve physician behaviour and patient outcome. They can have positive effects, but they can also have negative effects. Their effects must be tested by research.MethodsIn a randomized controlled trial, 103 psychiatrists in private practice were either provided with the WHO depression guideline only (information group), or provided with the WHO depression guideline and trained for one day in this guideline (intervention group), or left uninformed (control group). They then treated a total of 497 patients…

AdultMalemedicine.medical_specialtyEvidence-based practiceInservice TrainingMirtazapineMianserinAntidepressive Agents TricyclicWorld Health Organizationlaw.invention03 medical and health sciences0302 clinical medicineContinuing medical educationRandomized controlled triallawGermanymedicineHumans030212 general & internal medicinePsychiatryAgedPsychiatryDepressive Disorderbusiness.industryEvidence-based medicineGuidelineMiddle AgedMental health030227 psychiatryPsychiatry and Mental healthAffectPrivate practicePractice Guidelines as TopicPhysical therapyFemalebusinessPsychosocialEuropean psychiatry : the journal of the Association of European Psychiatrists
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Anti-anhedonic actions of the novel serotonergic agent flibanserin, a potential rapidly-acting antidepressant

1998

Chronic exposure to mild unpredictable stress has previously been found to depress the consumption of palatable sweet solutions and to block the formation of conditioned place preferences; these effects are reversed by chronic treatment with tricyclic or atypical antidepressant drugs. The present study was designed to evaluate the antidepressant-like activity in this model of flibaserin (BIMT-17), a novel serotonergic agent with 5-HT1A receptor agonist and 5-HT2 receptor antagonist properties. Two experiments were conducted, using rats (experiment 1) and mice (experiment 2). In experiment 1, decreases in sucrose intake were seen in rats exposed to chronic mild stress, but the effect was unr…

MaleAgonistSucrosemedicine.medical_specialtyQuinpirolemedicine.drug_classMotor ActivityMiceSerotonin AgentsQuinpiroleDopamine receptor D3Dopamine receptor D2Internal medicineSalicylamidesmedicineAnimalsRats WistarPharmacologyRacloprideFluoxetinebusiness.industryBody WeightFeeding BehaviorConditioned place preferenceRatsEndocrinologyRacloprideAntidepressive Agents Second-GenerationConditioning OperantDopamine AntagonistsFlibanserinBenzimidazolesbusinessStress Psychologicalmedicine.drugEuropean Journal of Pharmacology
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